The Scientific Committee needs to know your interest of participation with a specific paper, so contact the Scientific Committee indicating the tentative title.
According to the Committee´s decision, your paper will be included either in an oral or poster session and you will have proper indications for your presentation.
Submission of abstracts
Delegates who wish to propose papers for presentation at the Congress should edit abstracts in Office Word, format Arial 12, with symmetric margins 2,5 cm and send them as an attachment by email before January 30th, 2013, addressed to:
You must specify in the Subject: ABSTRACT FOR HEMATOLOGY 2013.
The heading should have the following lines:
Title of the paper in blacks.
Authors in a separate paragraph: last name, initials of first name(s); underline the name of the person who will present the paper.
Institution or Organization: official name of the organization of each of the authors; mark with a digit to whom each one corresponds to. Finally, the City and Country of the organizations.
Email of the author presenting the paper.
The text, in one paragraph, will follow and shall include a brief introduction, methods, main results and most relevant conclusions. The text must not exceed 300 words.
For papers to be presented as conferences or as part of a symposium or round table, the Abstract must comply the same requirements and adapt the body of the abstract to the topic referred to.
Abstracts not fulfilling these requirements will not be accepted. Only the ones received by email will be admitted.
See the following example:
Roberts syndrome associated with immunodeficiency
Sánchez-Segura M,1 Marsán-Suárez V1, Macías-Abraham C1, de León-Ojeda N2, García-García A2, Toledo-Rodríguez I3, del Valle-Pérez L1, Socarrás-Ferrer BB1
1Instituto de Hematología e Inmunología. La Habana, Cuba.
2Hospital Pediátrico Docente “William Soler”. La Habana, Cuba.
3Facultad de Ciencias Médicas “Comandante Manuel Fajardo”. La Habana, Cuba.
Roberts syndrome is an extremely rare genetic disease of autosomal recessive. It is clinically characterized by pre and postnatal growth delaying, severe limb shortening, radial defects, oligodactyly and craniofacial anomalies caused by mutation in the ESCO2 gene. This mutation encodes an acetyltransferase involved in regulating cohesion of sister chromatids. To our knowledge, no deficit of the immunological system has been described in this syndrome. We present a case of a one-and-a-half year old boy with Roberts syndrome, recurrent infectious processes, some of them severe, since his first year of life. Immunological studies showed decreased levels of Ig A, decreased number of CD3 positive T lymphocytes and decreased CD4 positive; cells with normal B quantitation and opsonophagocytic function impairment were also observed. A combined immunodeficiency associated with defective phagocytosis was diagnosed. Identifying an immunodeficiency associated with this genetic syndrome suggests that it corresponds to a genetically heterogeneous disease. This also shows the usefulness of the immunological assessment in patients with genetic defects and recurrent infections.